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Dermatologist (skin), Ophthalmologist (eyes)
19 years of experience
Accepting new patients
Video profile


Education ?

Medical School Score
Northeastern Ohio Universities (1993)

Awards & Distinctions ?

Patients' Choice Award (2010 - 2011, 2013 - 2015)
Compassionate Doctor Recognition (2010, 2013 - 2015)
Top 10 Doctor - City (2014)
Sarasota, FL
On-Time Doctor Award (2015)
University of Cincinnatti
Instructor Clin
University Of Cincinnati College Of Medi
American Society for Dermatologic Surgery
American Board of Dermatology
American Academy of Ophthalmology

Affiliations ?

Dr. Neff is affiliated with 6 hospitals.

Hospital Affiliations



  • University Hospital, Inc
    234 Goodman St, Cincinnati, OH 45267
    Top 25%
  • Adams County Hospital
    210 N Wilson Dr, West Union, OH 45693
    Top 25%
  • Sarasota Memorial Hospital
    1700 S Tamiami Trl, Sarasota, FL 34239
    Top 25%
  • Fort Hamilton Hospital
    630 Eaton Ave, Hamilton, OH 45013
    Top 50%
  • Anne Bates Leach Eye Hospital
  • Pht Jackson Memorial Hospital
  • Publications & Research

    Dr. Neff has contributed to 7 publications.
    Title Papular Amyloidosis Limited to the Ears.
    Date May 2010
    Journal Journal of the American Academy of Dermatology
    Title Optic Canal Decompression: a Cadaveric Study of the Effects of Surgery.
    Date September 2007
    Journal Ophthalmic Plastic and Reconstructive Surgery

    PURPOSE: To simulate a transphenoidal medial optic canal decompression and determine the anatomic effect on the optic nerve. METHODS: A medial optic canal decompression was performed on 5 cadaveric optic canals within 12 hours of death. Two canals were decompressed under direct visualization and 3 were decompressed by a transphenoidal endoscopic approach. The optic canal was subsequently removed en bloc, beginning at the annulus of Zinn and extending to the optic chiasm. Each specimen was processed and examined grossly. Serial coronal step sections of the entire length of the intracanalicular optic nerve were assessed histologically. RESULTS: Microscopic examination of the intracanalicular portion of optic nerve revealed incision in an extraocular muscle at the annulus, incomplete bone removal, fraying of the dural sheath, incomplete dural/arachnoid release, and incision in the pia and optic nerve. CONCLUSIONS: Transphenoidal medial wall decompression of the optic nerve canal with dural sheath opening may induce physical damage to the nerve. Any hypothetical value in dural-arachnoid sheath opening must be weighed against the potential for harm to the optic nerve caused by the surgical intervention.

    Title Lacrimal Gland Ductal Cyst Abscess.
    Date July 2001
    Journal Ophthalmic Plastic and Reconstructive Surgery

    PURPOSE: To describe a case of lacrimal gland ductal cyst complicated by secondary infection. METHODS: Case report. RESULTS: A 51-year-old woman presented acutely with an enlarging, painful mass in the superotemporal fornix. Clinical examination, echography, and surgical evaluation revealed a lacrimal gland ductal cyst with abscess formation. The lacrimal gland cyst was treated with oral antibiotics in combination with incision, drainage, and marsupialization. CONCLUSIONS: Lacrimal gland ductal cysts are rare but must be considered in the differential diagnosis of lacrimal gland and upper eyelid mass lesions. Typically, lacrimal gland ductal cysts develop after chronic inflammation, infection, or trauma. We describe a patient who presented acutely with a lacrimal gland ductal cyst associated with a rare complication of abscess formation.

    Title Posterior Fixation Suture Augmentation of Full-tendon Vertical Rectus Muscle Transposition for Abducens Palsy.
    Date September 2000
    Journal Journal of Neuro-ophthalmology : the Official Journal of the North American Neuro-ophthalmology Society

    OBJECTIVE: To evaluate the effect of augmenting full-tendon vertical rectus transpositions with posterior fixation sutures in patients with complete or near-complete lateral rectus palsy. METHODS: Transposition of the vertical recti to the lateral rectus muscle was performed in seven patients with unilateral lateral rectus palsy (the mean angle of preoperative horizontal deviation in primary gaze was 36.7 prism diopters (delta); range, 25-62delta of esotropia). A posterior fixation suture of 5.0 Mersilene (Ethicon, Somerville, NJ) was placed in sclera (14-16 mm posterior to the limbus) adjacent to the lateral rectus and incorporated 1/3 belly width of each transposed vertical rectus muscle. RESULTS: The mean angle of postoperative horizontal deviation in primary gaze was 7.1delta (range, 0-20delta). The mean change in primary-position horizontal deviation postoperatively was 41.2delta (range, 37-72delta). Four patients were able to fuse without prism in primary gaze; three patients were orthophoric and one patient had a consecutive intermittent exotropia. The remaining three patients required prism correction to neutralize the postoperative gaze deviation. All patients had improvement in abduction. Mild limitation of adduction was noted in three patients (range, -0.5 to -2.0). CONCLUSIONS: Augmenting full vertical rectus muscle transpositions with posterior fixation sutures improves the abducting effect of surgery without significant limitation of adduction.

    Title Giant Cell Arteritis Update.
    Date April 2000
    Journal Seminars in Ophthalmology

    Giant cell arteritis (GCA) is a systemic disorder characterized by vasculitis involving medium and large arteries. GCA is an ophthalmologic and systemic emergency, because devastating consequences, such as blindness and death, can occur without treatment. With prompt recognition and appropriate treatment, complications are almost always preventable. The prognosis for recovery of vision already lost because of GCA is usually poor, underscoring the absolute necessity of immediate intervention. Knowledge of the various manifestations of GCA is essential for recognition of this disorder early enough to prevent complications. This article highlights the important points regarding diagnosis, management, and complications associated with treatment of GCA.

    Title Intracameral Muramyl Dipeptide-induced Paracellular Permeability Associated with Decreased Glutamate Transporter and Gamma -glutamyltranspeptidase Activities.
    Date June 1999
    Journal Experimental Eye Research

    Muramyl dipeptide (MDP) (N -acetylmuramyl- L -alanyl- D - isoglutamine) was injected intracamerally to test if MDP applied to the aqueous side of the blood-aqueous barrier would increase paracellular permeability in association with diminished uptake of glutamate. The symptoms of anterior uveitis, i.e., increase in vascular dilatation, could be detected as early as 30 min post MDP injection while aqueous protein concentration did not increase at this time suggesting an initial dissociation between the circulatory and epithelial barrier responses. However, at 45 min, the aqueous protein concentration increased 10-fold (201+/-174 to 2094+/-1835 micrograms ml-1;P<0.001) rising progressively to 20-fold above the control eye at 60 min post injection (254+/-194 vs. 5038+/-2514 micrograms ml-1;P<0.001). Epithelial cell barrier paracellular permeability increased at 45 min as evidenced by the enhanced efflux of radiolabelled L -glucose out of the aqueous (8% and 13% faster than control at 45 and 60 min post MDP injection, respectively), coinciding with the accelerated protein influx. A near 50% reduction in efflux of both radiolabelled glutamate and D -aspartate was consistent with reduced glutamate uptake by the transport system X-AG. In addition, a 24% decline in aqueous glutamate, but not aspartate, was detected in the aqueous of the MDP-treated eyes in association with a 54% decrease in iris/ciliary body gamma-glutamyltranspeptidase activity consistent with reduced de novo glutamate formation from glutamine. The aqueous of MDP injected eyes also had 6-fold and 34-fold higher prostaglandin E2and F2alphaconcentrations, respectively (P</=0.03) as well as reduced AH bicarbonate concentration. These results suggest that increased paracellular permeability is associated with diminished gamma-glutamyltranspepidase-mediated glutamate production, X-AGtransport activity, and cellular acidosis in the MDP-induced prostaglandin-mediated inflammation.

    Title Treatment Strategies in Mucous Membrane Pemphigoid.
    Journal Therapeutics and Clinical Risk Management

    Mucous membrane pemphigoid (MMP) is an autoimmune blistering disorder that is characterized by subepithelial bullae. Various basement membrane zone components have been identified as targets of autoantibodies in MMP. Considerable variability exists in the clinical presentation of MMP. Mucous membranes that may be involved include the oral cavity, conjunctiva, nasopharynx, larynx, esophagus, genitourinary tract, and anus. A multidisciplinary approach is essential in the management of MMP. Early recognition of this disorder and treatment may decrease disease-related complications. The choice of agents for treatment of MMP is based upon the sites of involvement, clinical severity, and disease progression. For more severe disease, or with rapid progression, systemic corticosteroids are the agents of choice for initial treatment, combined with steroid-sparing agents for long-term maintenance. Due to the rarity of this disease, large controlled studies comparing the efficacy of various agents are lacking.

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