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Internist, Critical Care Specialist, Pulmonologist (lungs)
7 years of experience
Accepting new patients


Education ?

Medical School Score Rankings
University of Wisconsin at Madison (2004)
Top 25%

Awards & Distinctions ?

American Board of Internal Medicine

Affiliations ?

Dr. Sonetti is affiliated with 4 hospitals.

Hospital Affiliations



  • University of Wisconsin Hospital and Clinics
    Pulmonary Disease
    600 Highland Ave, Madison, WI 53792
  • University Health System
  • Audie L. Murphy Veterans Hospital
  • University Hospital
  • Publications & Research

    Dr. Sonetti has contributed to 1 publication.
    Title Role of Lung Inflammatory Mediators As a Cause of Exercise-induced Arterial Hypoxemia in Young Athletes.
    Date December 2002
    Journal Journal of Applied Physiology (bethesda, Md. : 1985)

    We examined whether lung inflammatory mediators are increased during exercise and whether pharmacological blockade can prevent exercise-induced arterial hypoxemia (EIAH) in young athletes. Seventeen healthy athletes (9 men, 8 women; age 23 +/- 3 yr) with varying degrees of EIAH completed maximal incremental treadmill exercise tests after administration of fexofenadine, zileuton, and nedocromil sodium or placebo in a randomized double-blind crossover study. Lung function, arterial blood gases, and inflammatory metabolites in plasma, urine, and induced sputum were assessed. Drug administration did not improve EIAH or gas exchange during exercise. At maximal exercise, oxygen saturation fell to 91.4 +/- 2.6% (drug trial) and 91.9 +/- 2.1% (placebo trial) and alveolar-arterial oxygen difference widened to 28.1 +/- 6.3 Torr (drug trial) and 29.3 +/- 5.7 Torr (placebo trial). Oxygen consumption, ventilation, and other exercise variables were similarly unaffected by drug treatment. Although plasma histamine increased with exercise, values did not differ between trials, and urinary leukotriene E(4) and 11beta-prostaglandin F(2alpha) levels were unchanged after exercise. Postexercise sputum revealed no significant changes in markers of inflammation. These results demonstrate that EIAH in young athletes is not attenuated with acute administration of drugs targeting histamine and bioactive lipids. We conclude that airway inflammation is of insufficient magnitude to cause impairments in gas exchange and does not appear to be linked to EIAH in healthy young athletes.

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