Internists, Cardiologist (heart)
28 years of experience

Accepting new patients
2015 Galloping Hill Rd
Kenilworth, NJ 07033
908-740-7405
Locations and availability (9)

Education ?

Medical School Score
Drexel University (1982)
  • Currently 2 of 4 apples

Affiliations ?

Dr. Berman is affiliated with 1 hospitals.

Hospital Affilations

Score

Rankings

  • Temple University Hospital
    Cardiology
    3401 N Broad St, Philadelphia, PA 19140
    • Currently 4 of 4 crosses
    Top 25%
  • Publications & Research

    Dr. Berman has contributed to 3 publications.
    Title Factors Affecting Survival After Heart Transplantation: Comparison of Pre- and Post-1999 Listing Protocols.
    Date June 2006
    Journal The Journal of Heart and Lung Transplantation : the Official Publication of the International Society for Heart Transplantation
    Excerpt

    BACKGROUND: Listing status for heart transplantation (Tx) patients was changed in 1999 from Status 1 and 2 to Status 1A, 1B and 2. Because the selection process was modified in favor of seriously ill patients, it was not clear whether this change would affect survival or other aspects of transplant management. METHODS: We examined outcomes in 551 patients transplanted at our institution between 1986 and 2002 (pre-1999: n = 419; post-1999: n = 132) to determine the effects of change in listing protocol on transplant outcome. RESULTS: Using Cox proportional hazard analysis, survival was not different between pre-1999 (pre) and post-1999 (post). Overall waiting-list times were longer post-1999 (pre: 134 +/- 10.5 days, post: 172 +/- 15.6 days; p = 0.044), and were longer post-1999 for blood groups A (177 vs 123 days), B (96 vs 84 days) and O (229 vs 172 days), but were shorter post for blood group AB (42 vs 68 days). Survival was not affected by age (pre: 53.7 +/- 0.52 years, post: 53.1 +/- 1.04 years; hazard ratio [HR] 1.00; 95% confidence interval [CI] 0.996 to 1.023; p = 0.181), male gender (HR 1.132; 95% CI 0.822 to 1.56; p = 0.447) or waiting-list time. Serum creatinine was similar between the 2 groups (pre: 1.25 +/- 0.02, post: 1.26 +/- 0.04; p = 0.794), whereas pulmonary artery (PA) diastolic pressure was increased post-1999 (pre: 24.9 +/- 0.46, post: 27.0 +/- 0.74; p = 0.023). Survival was not affected by PA pressure (HR 1.00; 95% CI 0.986 to 1.014; p = 0.976), but an elevated pre-transplant creatinine reduced survival (HR 1.484; 95% CI 1.139 to 1.933; p = 0.003). CONCLUSIONS: The change in listing status implemented in 1999 caused an increase in wait times for patients with blood types A, B and O, and shortened wait time for type AB; however, no differences occurred in overall post-transplant survival after the change in listing protocol. Age, gender and PA pressure had no effect on survival in either time period, whereas pre-transplant serum creatinine decreased survival in both patient groups.

    Title Effects of Sample Volume Location, Imaging View, Heart Rate and Age on Tricuspid Velocimetry in Normal Subjects.
    Date May 1990
    Journal The American Journal of Cardiology
    Excerpt

    The effects of imaging view and sample volume location on tricuspid velocimetry and of heart rate and aging on mitral and tricuspid inflow were evaluated in 41 normal subjects aged 20 to 76 years. Pulsed Doppler recordings were obtained in the parasternal short-axis, right ventricular inflow and apical 4-chamber views at the level of the tricuspid and mitral anuli and 1 cm caudad and 1 cm cephalad to the tricuspid anulus in the 4-chamber apical view. The right ventricular filling pattern was not affected by imaging view. However, placement of the sample volume 1 cm cephalad resulted in a 16% reduction (p less than 0.01) in early velocity with a 9% increase in atrial filling fraction. Conversely, late velocity was 11% higher (p less than 0.05) at the anular level versus the other locations. Right and left ventricular filling velocities were modestly related (r = 0.50 to 0.63). Relations between age and tricuspid late velocity, velocity ratio and atrial filling fraction were weaker (r = 0.34 to 0.47; all p less than 0.05) than those between age and mitral variables (r = 0.59 to 0.74. Also, aging had a greater effect on mitral than tricuspid late velocity (i.e., a steeper slope; p less than 0.01). Tricuspid late velocity and atrial filling fraction were each modestly inversely related to RR interval (r = -0.48, r = -0.54; p less than 0.01). Thus, tricuspid velocity is affected by sample volume location, aging and heart rate, but not imaging view. Sample volume location, heart rate and age should be considered when evaluating right ventricular inflow parameters.

    Title Prevalence of Multivalvular Regurgitation in Athletes.
    Date August 1989
    Journal The American Journal of Cardiology
    Excerpt

    To assess the effects of exercise training on the prevalence of valvular regurgitation, 2-dimensional echocardiography and Doppler flow mapping were performed in 45 athletes and 26 sedentary control subjects of similar age and sex. Mitral, tricuspid, aortic and pulmonic regurgitations were sought in all possible views and mitral and tricuspid flow velocities were recorded. Mitral and tricuspid anulus diameters and the maximal areas of regurgitant flow were planimetered. Regurgitation of at least one of the cardiac valves was found in 91% of athletes but in only 38% of control subjects (p less than 0.001). Mitral and tricuspid regurgitation occurred more commonly in athletes than in control subjects (mitral 69 vs 27%; tricuspid 76 vs 15%). The prevalence of aortic and pulmonic regurgitation was similar. Although athletes and sedentary normal subjects differed with respect to heart rate, right and left ventricular filling patterns and tricuspid and mitral anulus diameters, none of these variables was related to the presence or severity of regurgitation. Thus, exercise training is associated with an increased prevalence of mitral and tricuspid regurgitation and altered ventricular inflow patterns. The mechanism of these findings is unclear. Multivalvular regurgitation is common in athletes and does not imply structural valvular abnormalities.

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