Internists, Cardiologist (heart)
18 years of experience

Accepting new patients
Alaska Heart Institute
3841 Piper St
Ste T100
Anchorage, AK 99508
Locations and availability (6)

Education ?

Medical School
Vrije Universiteit Brussels (1992)
Foreign school

Awards & Distinctions ?

Patients' Choice Award (2008 - 2009)
Compassionate Doctor Recognition (2009)
Top Ten Doctors (2012)
Cardiology, Tudor Municipal Campus, Anchorage, AK

Affiliations ?

Dr. Strobel is affiliated with 8 hospitals.

Hospital Affilations



  • Alaska Regional Hospital
    2801 Debarr Rd, Anchorage, AK 99508
    • Currently 3 of 4 crosses
    Top 50%
  • Valley Hospital
    2500 S Woodworth Loop, Palmer, AK 99645
    • Currently 3 of 4 crosses
    Top 50%
  • Central Peninsula General Hospital
    250 Hospital Pl, Soldotna, AK 99669
    • Currently 2 of 4 crosses
  • Providence Alaska Medical Center
    PO Box 196604, Anchorage, AK 99519
    • Currently 2 of 4 crosses
  • Providence Seward Wesley Care Center
    PO Box 365, Seward, AK 99664
  • Providence Valdez Medical Center
    Valdez, AK 99686
  • Alaska Heart Institute, LLC
  • Providence Extended Care Center
    4900 Eagle St, Anchorage, AK 99503
  • Publications & Research

    Dr. Strobel has contributed to 3 publications.
    Title Greater Pathogen Burden but Not Elevated C-reactive Protein Increases the Risk of Clinical Restenosis After Percutaneous Coronary Intervention.
    Date September 2002
    Journal American Heart Journal

    BACKGROUND: Restenosis after percutaneous coronary intervention (PCI) constitutes a serious complication in the treatment of cardiovascular disease, but known risk factors do not fully account for the observed restenosis risk. Preliminary studies of infection or inflammation in restenosis report varied results. We tested whether C-reactive protein (CRP) or pathogen burden (seropositivity to 0, 1, 2, or 3 pathogens, of Chlamydia pneumoniae [Cpn], cytomegalovirus [CMV], or Helicobacter pylori [Hpy]) predict clinical restenosis after percutaneous coronary intervention (PCI). METHODS: Blood samples were collected from 415 patients undergoing PCI, and levels of plasma CRP and antibodies to Cpn, CMV, and Hpy were measured. The patient's medical history, demographics, and procedural data were recorded. Patient end points were determined for as long as 6 months as a means of evaluating the incidence of clinical restenosis and major adverse cardiac events. RESULTS: The average patient age was 62 years, and 80% of patients were male. Fifty-eight patients (14%) experienced clinical restenosis, whereas 17 patients (4%) died or had an acute myocardial infarction. After adjusting for 19 possible predictors, we found the pathogen burden (P-trend =.04, adjusted odds ratio [OR] 1.5 per number of pathogens) and minimum luminal diameter (P =.003, OR 1.8 per mm decrease) to be significant predictors of clinical restenosis. Male sex was a nonsignificant predictor of restenosis (P =.06, OR 2.2), but CRP was not significant after adjustment (P-trend =.10, OR 0.73 per tertile). CONCLUSION: Pathogen burden was associated with clinical coronary restenosis, an association that deserves further exploration and evaluation. CRP, a marker of inflammation, was not associated with an increased risk of restenosis.

    Title Successful Pediatric Stenting of a Nonthrombotic Coronary Occlusion As a Complication of Radiofrequency Catheter Ablation.
    Date May 2002
    Journal Pacing and Clinical Electrophysiology : Pace

    This is a case of a right coronary artery occlusion complicating a RF catheter ablation of a posteroseptal accessory connection in an 8-year-old boy. After multiple balloon angioplasty attempts in the occluded vessel, only transient patency was achieved. The occlusion was successfully treated with placement of an intracoronary stent.

    Title Sesquiflutter.
    Date November 1999
    Journal Journal of Cardiovascular Electrophysiology

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