Surgical Specialist, Urologists
28 years of experience
Video profile
Accepting new patients
Bryn Mawr
Center For Urologic Care
245 S Bryn Mawr Ave
Bryn Mawr, PA 19010
610-525-2515
Locations and availability (2)

Education ?

Medical School Score Rankings
Columbia University (1982)
  • Currently 4 of 4 apples
Top 25%

Awards & Distinctions ?

Associations
American Society for Reproductive Medicine
American Board of Urology
American Urological Association
American College of Surgeons

Affiliations ?

Dr. Bernstein is affiliated with 20 hospitals.

Hospital Affilations

Score

Rankings

  • Main Line Hospital - Bryn Mawr
    Urology
    130 S Bryn Mawr Ave, Bryn Mawr, PA 19010
    • Currently 4 of 4 crosses
    Top 25%
  • Bryn Mawr Rehabilitation Hospital
    414 Paoli Pike, Malvern, PA 19355
    • Currently 4 of 4 crosses
    Top 25%
  • Riddle Memorial Hospital
    1068 W Baltimore Pike, Media, PA 19063
    • Currently 3 of 4 crosses
    Top 50%
  • Mercy Fitzgerald Hospital
    1400 Lansdowne Ave, Darby, PA 19023
    • Currently 2 of 4 crosses
  • Main Line Hospital Paoli
    255 W Lancaster Ave, Paoli, PA 19301
    • Currently 2 of 4 crosses
  • Main Line Hospital Lankenau
    Urology
    100 E Lancaster Ave, Wynnewood, PA 19096
    • Currently 1 of 4 crosses
  • Lankenau Medical Center - On staff since
  • -Paoli Hospital - On staff since 1995
  • -Lankenau Hospital - On staff since 1997
  • Paoli Mem Hosp, Paoli, Pa
  • -Bryn Mawr Rehabilitation Hosp - On staff since 1989
  • M L Health-Paoli Memorial Hospital
  • Lankenau Medical Center
  • -Bryn Mawr Hospital - On staff since 1988
  • Bryn Mawr Hospital
  • Mercy Community Hospital, Havertown, Pa
  • Bryn Mawr Hosp, Bryn Mawr, Pa
  • Lankenau Hosp, Wynnewood, Pa
  • M L Hospital Bryn Mawr Hospital
  • M L Hospital Lankenau
  • Publications & Research

    Dr. Bernstein has contributed to 4 publications.
    Title The Physiologic Basis of the Tur Syndrome.
    Date April 1989
    Journal The Journal of Surgical Research
    Excerpt

    To better assess the role of hyperammonemia versus hypoosmolarity versus hyponatremia in the TUR syndrome, we developed a rat model. Sprague-Dawley female rats received an intraperitoneal injection (250 cc/kg body weight) of either 1.5% glycine, 2.0% glycine, 2.0% glycine plus 1.5% mannitol, 3.0% mannitol, 5.0% mannitol, or 2.0% glycine plus 0.25% saline. Arterial blood samples were obtained prior to injection, at 2, 8, 16, and 24 hr and analyzed for osmolarity, sodium, and ammonia. Those animals receiving 2.0% glycine, 2.0% glycine plus 1.5% mannitol, and 5.0% mannitol all died within 24 hr with lethargy, convulsions, and coma. Hyponatremia developed in all animals; death, however, occurred only when the sodium concentration declined to 90-95 meq/dl. Mannitol maintained serum osmolarity but did not prevent coma and death. Including 0.25% saline in the initial injection, or an iv injection of 5.0% saline delayed 8 hr achieved 100% survival. Ammonia concentrations increased 15-fold by 8 hr in groups receiving 2.0% glycine; it rapidly decreased to near normal by 24 hr. Decreasing the rise in ammonia by 50% with iv arginine had no effect on survival. Our results suggest that hyponatremia rather than hyperammonemia or hypoosmolarity accounts for the major morbidity and mortality secondary to the TUR syndrome.

    Title Ureteropelvic Junction Obstruction in the Neonate.
    Date November 1988
    Journal The Journal of Urology
    Excerpt

    The widespread use of maternal ultrasonography has resulted in an increasing number of asymptomatic newborns being diagnosed with hydronephrosis. A total of 89 neonates with hydronephrosis secondary to ureteropelvic junction obstruction have been referred for evaluation and treatment during the last 6 years. In the majority the condition was diagnosed readily with ultrasonography, voiding cystography and excretory urography. Twenty-five per cent underwent more quantitative assessment using nuclear renal imaging. Only 2 patients (8 per cent) had a percutaneous study. Early reconstruction was performed in 75 per cent of the patients and it proved to be safe and effective. Twenty-three neonates were followed nonoperatively. One patient (4 per cent) demonstrated clinical deterioration necessitating later surgical intervention. Careful surveillance is mandatory in all infants followed nonoperatively.

    Title Congenital Progressive Hydronephrosis in Mice: a New Recessive Mutation.
    Date November 1988
    Journal The Journal of Urology
    Excerpt

    We describe a new autosomal recessive mutation, congenital progressive hydronephrosis (cph), that arose in the C57BL/6J inbred mouse strain. The clinical, histopathological, biochemical and radiographic characteristics, and the genetic linkage of this new mutation are discussed. Our studies indicate that the homozygous mutant mice have progressive bilateral upper urinary tract obstruction leading to azotemia and death of renal failure. The anatomical site of obstruction appears to be at the level of the ureteropelvic junction. Genetic mapping studies have localized the cph gene to the distal half of chromosome 15. The cph mouse strain provides a reproducible model for analysis of the onset and development of obstructive uropathic conditions in the neonatal period.

    Title Transurethral Incisions and Resections Under Local Anaesthesia.
    Date November 1987
    Journal British Journal of Urology

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