Neurologist (brain, nervous system)
18 years of experience
Video profile
Accepting new patients
Oak Lawn
5303 Harry Hines Blvd
Dallas, TX 75390
214-590-5218
Locations and availability (2)

Education ?

Medical School
Smt. N.H.L. Municipal Medical College (1992)
Foreign school

Awards & Distinctions ?

Awards  
Trephined Cranium Teaching Award
Achievement in Clinical Science&Humanism in the Practice of Medicine (1997)
Trephined Cranium Teaching Award (2007)
Outstanding Resident (1997)
Senior Resident of the Year (2000)
Trephined Cranium Teaching Award (2003)
Associations
American Board of Psychiatry and Neurology
American Association of Neuromuscular and Electrodiagnostic Medicine

Affiliations ?

Dr. Trivedi is affiliated with 7 hospitals.

Hospital Affilations

Score

Rankings

  • UT Southwestern University Hospital - Zale Lipshy
    5151 Harry Hines Blvd, Dallas, TX 75235
    • Currently 4 of 4 crosses
    Top 25%
  • UT Southwestern University Hospital - St. Paul
    5909 Harry Hines Blvd, Dallas, TX 75235
    • Currently 4 of 4 crosses
    Top 25%
  • Parkland Health & Hospital System
    5201 Harry Hines Blvd, Dallas, TX 75235
    • Currently 1 of 4 crosses
  • St Paul Hospital
  • Dallas County Hospital District
  • UT Southwestern St Paul Hospital
  • UT Southwestern Zale Lipshy Hospital
  • Publications & Research

    Dr. Trivedi has contributed to 8 publications.
    Title Huntington Chorea Presenting with Motor Neuron Disease.
    Date July 2011
    Journal Archives of Neurology
    Excerpt

    There have been a few case reports of motor neuron disease in association with Huntington disease (HD).

    Title Is Female Sex an Independent Risk Factor for Perioperative Transfusion in Coronary Artery Bypass Graft Surgery?
    Date April 2011
    Journal Journal of the American College of Surgeons
    Excerpt

    Perioperative red blood cell transfusion is a risk factor for postoperative mortality and morbidity in coronary artery bypass grafting (CABG). Females have a higher risk of red blood cell transfusion, but few previous studies have accounted for preoperative hematocrit and female sex together as risk factors for red blood cell transfusion. We evaluated female sex as an independent risk factor for red blood cell transfusion in CABG, while accounting for hematocrit.

    Title Denture Cream: an Unusual Source of Excess Zinc, Leading to Hypocupremia and Neurologic Disease.
    Date September 2008
    Journal Neurology
    Excerpt

    BACKGROUND: Chronic, excess zinc intake can result in copper deficiency and profound neurologic disease. However, when hyperzincemia is identified, the source often remains elusive. We identified four patients, one previously reported, with various neurologic abnormalities in the setting of hypocupremia and hyperzincemia. Each of these patients wore dentures and used very large amounts of denture cream chronically. OBJECTIVE: To determine zinc concentration in the denture creams used by the patients as a possible source of excess zinc ingestion. METHODS: Detailed clinical and laboratory data for each patient were compiled. Tubes of denture adhesives were analyzed for zinc content using dynamic reaction cell-inductively coupled plasma-mass spectrometry. Patients received copper supplementation. Copper and zinc levels were obtained post-treatment at varying intervals. RESULTS: Zinc concentrations ranging from about 17,000 to 34,000 mug/g were identified in Fixodent and Poli-Grip denture creams. Serum zinc levels improved in three patients following cessation of denture cream use. Copper supplementation resulted in mild neurologic improvement in two patients who stopped using denture cream. No alternative source of excess zinc ingestion or explanation for hypocupremia was identified. CONCLUSION: Denture cream contains zinc, and chronic excessive use may result in hypocupremia and serious neurologic disease.

    Title Painful Peripheral Neuropathy and Its Nonsurgical Treatment.
    Date August 2004
    Journal Muscle & Nerve
    Excerpt

    Treatment of neuropathic pain is the primary focus of management for many patients with painful peripheral neuropathy. Antidepressants and anticonvulsants are the two pharmacological classes most widely studied and represent first-line agents in the management of neuropathic pain. The number of pharmacological agents that have demonstrated effectiveness for neuropathic pain continues to expand. In the current review, we summarize data from randomized, controlled pharmacological trials in painful peripheral neuropathies. Although neuropathic pain management remains challenging because the response to therapy varies considerably between patients, and pain relief is rarely complete, a majority of patients can benefit from monotherapy using a well-chosen agent or polypharmacy that combines medications with different mechanisms of action.

    Title Transverse Myelitis in Systemic Sclerosis.
    Date February 2004
    Journal Archives of Neurology
    Excerpt

    BACKGROUND: Neurological involvement occurs rarely with systemic sclerosis (SSc). Only a few cases of transverse myelopathy have been reported in the setting of SSc. OBJECTIVE: To describe a patient with SSc who developed transverse myelitis that improved during a course of immunosuppression. RESULTS: A 30-year-old woman with SSc presented with subacute onset of bilateral lower extremity weakness and numbness. Results of magnetic resonance imaging and cerebrospinal fluid studies supported a diagnosis of transverse myelitis. The patient responded favorably to a course of corticosteroids and cyclophosphamide. No overlapping autoimmune disorders were evident. Clinical follow-up showed significant recovery, with resolution of radiological abnormalities. CONCLUSION: Transverse myelitis can occur as a rare manifestation of SSc and may respond favorably to immunosuppressive therapy.

    Title Adult Polyglucosan Body Disease Associated with Lewy Bodies and Tremor.
    Date June 2003
    Journal Archives of Neurology
    Excerpt

    BACKGROUND: Adult polyglucosan body disease (PGBD) is rare and typically presents with upper and lower motor neuron involvement and neurogenic bladder. Extrapyramidal features are unusual in PGBD and are presumed secondary to widespread pathology that includes the basal ganglia. There are no prior reports of Lewy bodies in PGBD. OBJECTIVE: To report a unique finding of Lewy bodies in a patient with PGBD. REPORT OF A CASE A 46-year-old woman initially presented with a 4-year history of resting tremor. The tremor responded to levodopa therapy. Several months later, she developed upper and lower motor neuron involvement and other clinical features of PGBD. A sural nerve biopsy specimen revealed intra-axonal polyglucosan bodies that confirmed the clinical diagnosis. Bulbar and limb weakness progressed, and she developed dementia. She died 6 years after onset. At autopsy, extensive polyglucosan body formation was found in many regions of the central nervous system. In addition, numerous alpha-synuclein staining Lewy bodies were observed in the substantia nigra, accompanied by marked neuron depopulation. CONCLUSIONS: To our knowledge, this is the first report of adult PGBD associated with Lewy bodies and levodopa-responsive tremor. Although polyglucosan bodies were seen in substantia nigra, it is most likely that our patient had coexisting Parkinson disease.

    Title Primary Lateral Sclerosis: Clinical and Laboratory Features in 25 Patients.
    Date
    Journal Journal of Clinical Neuromuscular Disease
    Excerpt

    OBJECTIVE:: The objective of this study was to characterize the clinical features and natural history of primary lateral sclerosis (PLS). BACKGROUND:: PLS is a motor neuron disorder defined by corticospinal and corticobulbar tract dysfunction without clinically significant lower motor neuron involvement. METHODS:: We collected data from 25 patients with PLS seen in 2 academic neurology departments over a 5-year period. RESULTS:: The PLS population represented approximately 3% of acquired motor neuron disease cases seen during that period. Twenty-three patients (92%) presented with lower limb weakness, spasticity, or difficulty with ambulation. None presented with upper limb symptoms. Eleven patients (44%) developed bulbar symptoms. All patients had hyperreflexia and increased muscle tone. Muscle weakness was observed in 15 patients (60%) and tended to be mild and asymmetric. Needle electromyography (EMG) was normal or showed only fasciculations in 15 patients (60%); 10 patients had features of mild active denervation, consisting of fibrillation or positive sharp wave potentials, but the extent of these findings did not satisfy World Federation of Neurology electrophysiological criteria for the diagnosis of amyotrophic lateral sclerosis. Fourteen patients (52%) continued independent ambulation. Of the 10 patients with active denervation on EMG, 6 (60%) required a walker, scooter, or wheelchair at a mean follow up of 6.2 years. There were no fatalities over the 5-year period. CONCLUSIONS:: Our experience supports the observation that PLS progresses more slowly than other forms of acquired motor neuron disease, particularly amyotrophic lateral sclerosis. Follow-up data suggest that patients with active denervation changes develop greater disability.

    Title Von Hippel-lindau Disease Associated with Thymoma and Myasthenia Gravis.
    Date
    Journal Journal of Clinical Neuromuscular Disease
    Excerpt

    Von Hippel-Lindau (VHL) disease is an autosomal-dominant disorder characterized by central nervous system hemangioblastomas, retinal angioma, and renal cell carcinoma. Thymoma and autoimmune neurologic disorders have not been reported in association with VHL disease. We report a unique concurrence of antibody-positive myasthenia gravis and thymoma in a patient with VHL disease. Although this may be coincidental, a possible genetic link between thymoma and VHL is described.


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