Neurologist (brain, nervous system)
7 years of experience

Accepting new patients
Westwood
300 Ucla Medical Plz
Suite B200
Los Angeles, CA 90095
310-794-1195
Locations and availability (3)

Education ?

Medical School
Teheran University Of Medical Sciences And Health Services (2003)

Awards & Distinctions ?

Associations
American Board of Psychiatry and Neurology

Affiliations ?

Dr. Rafiei is affiliated with 4 hospitals.

Hospital Affilations

Score

Rankings

  • UCLA Medical Center
    10833 Le Conte Ave, Los Angeles, CA 90095
    • Currently 4 of 4 crosses
    Top 25%
  • Santa Monica - UCLA Medical Center
    1250 16th St, Santa Monica, CA 90404
    • Currently 3 of 4 crosses
    Top 50%
  • University of California - Ronald Reagan UCLA Medical Center
    757 Westwood Plz, Los Angeles, CA 90095
  • Mattel Chldns Hosp. At Ucla
    10833 Le Conte Ave, Los Angeles, CA 90095
  • Publications & Research

    Dr. Rafiei has contributed to 5 publications.
    Title Warfarin-induced Skin Necrosis of the Eyelids.
    Date March 2007
    Journal Archives of Ophthalmology
    Title Bilateral Dacroadenitis Associated with Acute Crohn's Disease.
    Date March 2007
    Journal Eye (london, England)
    Title Retinal Fibrovascular Proliferation Associated with Nocardia Subretinal Abscess.
    Date November 2006
    Journal European Journal of Ophthalmology
    Excerpt

    PURPOSE: To report the development of extensive fibrovascular proliferation in association with Nocardia subretinal abscess. METHOD: Case report. RESULTS: Extensive retinal neovascularization with tractional retinal detachment developed soon after ocular involvement in a 61-year-old patient with systemic nocardiosis. Fundus fluorescein angiography showed extensive area of capillary nonperfusion and severe leakage from the neovascular complex. The Nocardia subretinal abscess responded to systemic antibiotics, and the retinal neovascularization and tractional retinal detachment stabilized after 3 months. CONCLUSIONS: Retinal ischemia and severe retinal neovascularization may complicate intraocular nocardiosis. The authors propose secondary retinal vasculitis as a contributing factor towards the development of retinal ischemia in this setting.

    Title Characteristics and Outcomes of Choroidal Neovascularization Occurring After Macular Hole Surgery.
    Date December 2004
    Journal Retina (philadelphia, Pa.)
    Excerpt

    PURPOSE: To study the characteristics and outcomes of choroidal neovascularization (CNV) developing after surgery for idiopathic macular holes. METHODS: Retrospective, noncomparative case series. Search of diagnostic and procedure databases of four participating institutes identified nine eyes of eight patients with CNV after surgery for idiopathic macular hole. Demographic, clinical, and fundus fluorescein angiographic data were abstracted from patients' medical records. RESULTS: Choroidal neovascularization developed 6 weeks to 30 months after macular hole surgery. The macular hole was closed in all cases. The CNV complex involved the foveal region in 9 (100%) eyes, was predominantly classic in 8 (89%) eyes, and was associated with moderate to severe leakage of fluorescein in 8 (89%) eyes. The CNV was crescent-shaped and spared the central area of retinal pigment epithelial mottling that corresponded to the site of the macular hole in 6 (67%) cases. Other fluorescein angiographic features included retinal pigment epitheliopathy in 6 (67%) eyes and presence of drusen in 7 (78%) eyes. The final visual acuity after a mean follow-up period of 11.2 months (range, 3-16 months) was 20/50 to 20/80 in 2 (22%) eyes, 20/100 to 20/200 in 1 (11%) eye, and less than 20/200 in 6 (67%) eyes. CONCLUSION: Choroidal neovascular complexes occurring after macular hole surgery tend to share certain characteristics, including foveal involvement, frequent sparing of the central area corresponding to the site of the closed macular hole, and generally poor visual outcomes. Therapeutic modalities including photodynamic therapy and surgical options may be considered in these patients.

    Title Right Sensory Alien Hand Phenomenon from a Left Pontine Hemorrhage.
    Date
    Journal Journal of Clinical Neurology (seoul, Korea)
    Excerpt

    Acute onset of a sensory alien hand phenomenon has been observed only from a supratentorial lesion involving the non-dominant hand, mostly from a right posterior cerebral artery infarction. A single acute vascular lesion resulting in a dominant hand sensory alien hand syndrome has not been previously documented.


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