Internists, Hematology Specialist, Oncology Specialist (cancer)
12 years of experience

Accepting new patients
Garden Acres Area
Center For Cancer & Blood
11805 South Fwy
Ste 201
Burleson, TX 76028
817-551-5312
Locations and availability (19)

Education ?

Medical School
Siddhartha Medical College (1998)
Foreign school

Awards & Distinctions ?

Awards  
Bridges to Excellence Recognition
Physician Office Systems Recognition (2011 - 2014)
Level II
NCQA Physician Practice Connections (2011 - 2014)
Associations
American Board of Internal Medicine
American Society of Clinical Oncology

Affiliations ?

Dr. Potluri is affiliated with 22 hospitals.

Hospital Affilations

Score

Rankings

  • Harris Methodist H E B
    Medical Oncology
    1600 Hospital Pkwy, Bedford, TX 76022
    • Currently 4 of 4 crosses
    Top 25%
  • Texas Health Harris Methodist Hospital Fort Worth
    Medical Oncology
    1301 Pennsylvania Ave, Fort Worth, TX 76104
    • Currently 4 of 4 crosses
    Top 25%
  • Texas Health Presbyterian Hospital Of Dallas
    Medical Oncology
    8200 Walnut Hill Ln, Dallas, TX 75231
    • Currently 4 of 4 crosses
    Top 25%
  • Texas Health Harris Methodist Hospital Southwest Fort Worth
    6100 Harris Pkwy, Fort Worth, TX 76132
    • Currently 4 of 4 crosses
    Top 25%
  • Baylor Medical Center at Southwest Fort Worth
    1400 8th Ave, Fort Worth, TX 76104
    • Currently 3 of 4 crosses
    Top 50%
  • Texas Health Harris Methodist Hospital Azle
    108 Denver Trl, Azle, TX 76020
    • Currently 3 of 4 crosses
    Top 50%
  • Texas Health Harris Methodist Hospital Cleburne
    201 Walls Dr, Cleburne, TX 76033
    • Currently 2 of 4 crosses
  • Jps Health Network
    1500 S Main St, Fort Worth, TX 76104
    • Currently 2 of 4 crosses
  • Baylor All Saints Medical Centers
    Medical Oncology
    1400 8th Ave, Fort Worth, TX 76104
    • Currently 2 of 4 crosses
  • Usmd Surgical Hospital Of Arlington
    801 W Interstate 20, Arlington, TX 76017
    • Currently 1 of 4 crosses
  • Plaza Medical Center
    900 8th Ave, Fort Worth, TX 76104
    • Currently 1 of 4 crosses
  • Terrell State Hospital
    1200 E Brin St, Terrell, TX 75160
  • Methodist Mansfield Medical Center
    2700 E Broad St, Mansfield, TX 76063
  • JPS Diagnostic and Surgery Hospital of Arlington
    4400 New York Ave, Arlington, TX 76018
  • Texas Health Cleburne
  • Harris Continued Care Hospital
    1301 Pennsylvania Ave, Fort Worth, TX 76104
  • Texas Health Fort Worth
  • Texas Health Southwest Fort Worth
  • TX Health Southwest Fw
  • Huguley Memorial Medical Center
    11801 South Fwy, Fort Worth, TX 76115
  • Harris Methodist - Springwood
    1608 Hospital Pkwy, Bedford, TX 76022
  • Harris Methodist-Fw
  • Publications & Research

    Dr. Potluri has contributed to 4 publications.
    Title Continuous Noninvasive Measurement of Pulsus Paradoxus Complements Medical Decision Making in Assessment of Acute Asthma Severity.
    Date October 2006
    Journal Chest
    Excerpt

    BACKGROUND: Pulsus paradoxus (PP) is a pathophysiologic parameter that is indicative of asthma severity. The ability of PP to categorize acutely asthmatic patients in accordance with the earlier National Asthma Education and Prevention Program (NAEPP) expert panel report 1 guidelines was determined. METHODS: An arterial tonometric BP monitor, which was interfaced to an analog-digital converter, executed a periodic amplitude analysis algorithm, which computed PP in real time. The PP measurement was compared to the criterion standard of emergency physicians in determining the hospital admission vs hospital discharge disposition following the NAEPP standardized treatment. Receiver operating characteristics (ROCs) were calculated, and the PP threshold, which maximized sensitivity and specificity, was identified. In a separate laboratory investigation, PP was induced in a healthy volunteer by inspiration through a fixed resistance. Plethysmographic waveform changes, induced by PP, were measured by a second analog-to-digital converter that was connected to a pulse oximeter. RESULTS: A total of 79 patients were enrolled in the study, of whom 63 met a priori inclusion criteria and had uninterrupted data acquisition. The mean PP for patients who were appropriately discharged from the hospital was 9.1 mm Hg (95% confidence interval [CI], 7.3 to 10.9 mm Hg) and differed from the PP of 17.6 mm Hg (95% CI, 13.5 to 21.8; p < 0.001) for patients admitted to the hospital/relapsed. The sensitivity and specificity for physician disposition were 0.83 and 0.89, respectively, and for PP values were 0.78 and 0.78, respectively. The Wilcoxon area under the ROC curve was 0.82 (95% CI, 0.64 to 0.99) following treatment. The risk ratio was 5.32 for hospital admission among patients with a PP of > 11.3 mm Hg. Changes in the photoplethysmography peak height were correlated to PP from the BP monitor by a regression line with a slope of 0.01 V/mm Hg. CONCLUSIONS: Continuous PP can aid in determining disposition among emergency department (ED) patients with acute asthma. ED physicians equipped with a PP monitor would be able to objectify the work of breathing and would more closely adhere to NAEPP guidelines. The possibility that a PP detection algorithm could reside in a pulse oximeter warrants further investigation.

    Title Chemotherapy with Taxanes in Breast Cancer During Pregnancy: Case Report and Review of the Literature.
    Date September 2006
    Journal Clinical Breast Cancer
    Excerpt

    Two patients with breast cancer received docetaxel-containing chemotherapy as adjuvant or neoadjuvant therapy during pregnancy. The first pregnant patient began neoadjuvant therapy with doxorubicin/cyclophosphamide at 14 weeks of gestation. After 4 cycles of doxorubicin/cyclophosphamide and surgery, she received adjuvant docetaxel for 4 cycles. The second patient began neoadjuvant therapy with doxorubicin/docetaxel at 14 weeks of gestation and received 6 cycles. The fetus of the first patient had hydrocephalus on ultrasound at 17 weeks of gestation (before docetaxel therapy) that persisted on serial follow-up ultrasounds and spontaneously regressed over several months after delivery. No fetal malformations were detected in the second fetus. These 2 cases add to the existing data on the use of taxanes during pregnancy. Although the data are limited with case reports, pregnant patients with cancer can be treated with chemotherapy including taxanes during the second and third trimesters without significant risks to the fetus. Taxanes should not be excluded, if indicated, in pregnant patients with cancer.

    Title Cloning and Characterization of Zebrafish Tbx1.
    Date July 2004
    Journal Gene Expression Patterns : Gep
    Excerpt

    Tbx1 is one of the genes within the DiGeorge Critical Region (DGCR) and has been recently identified as the critical gene for the cardiovascular anomalies in the DiGeorge mouse models. We have cloned, sequenced and analyzed the zebrafish (Danio rerio) tbx1 cDNA. It encodes a protein of 460 amino acids that shares 64% identity and 67% similarity with the human TBX1 orthologue at the amino acid level. Although maternal expression was detected by RT-PCR, only zygotic expression could be detected by whole-mount in situ hybridization. Expression of zebrafish tbx1 by whole-mount in situ hybridization was first detected at 40% epiboly, 5.0 hours post fertilization (hpf) in the dorsal blastoderm margin. Through the stage of embryonic shield formation, tbx1 expression is restricted to the hypoblast, in the region of cells fated to become head and lateral plate mesoderm and pharyngeal endoderm. At 18 hpf, when the heart tube is beginning to assemble, three domains of tbx1 expression can be seen: cardiac precursors, pharyngeal arch precursors and otic vesicle. These three domains will remain the sites of tbx1 expression to varying degrees through at least 72 hpf. By 51 hpf, tbx1 expression can be seen in the cardiac outflow tract, the ventricle and the atrium, although by 72 hpf cardiac expression is strongest in the cardiac outflow tract. This newly identified tbx1 expression pattern in cardiac regions other than the cardiac outflow tract offers a new insight into the role of the tbx1 transcription factor in cardiac development.

    Title The Role of Neural Crest During Cardiac Development in a Mouse Model of Digeorge Syndrome.
    Date December 2002
    Journal Developmental Biology
    Excerpt

    The velo-cardio-facial syndrome (VCFS)/DiGeorge syndrome (DGS) is a genetic disorder characterized by phenotypic abnormalities of the derivatives of the pharyngeal arches, including cardiac outflow tract defects. Neural crest cells play a major role in the development of the pharyngeal arches, and defects in these cells are likely responsible for the syndrome. Most patients are hemizygous for a 1.5- to 3.0-Mb region of 22q11, that is suspected to be critical for normal pharyngeal arch development. Mice hemizygous for a 1.5-Mb homologous region of chromosome 16 (Lgdel/+) exhibit conotruncal cardiac defects similar to those seen in affected VCFS/DGS patients. To investigate the role of Lgdel genes in neural crest development, we fate mapped neural crest cells in Lgdel/+ mice and we performed hemizygous neural crest-specific inactivation of Lgdel. Hemizygosity of the Lgdel region does not eliminate cardiac neural crest migration to the forming aortic arches. However, neural crest cells do not differentiate appropriately into smooth muscle in both fourth and sixth aortic arches and the affected aortic arch segments develop abnormally. Tissue-specific hemizygous inactivation of Lgdel genes in neural crest results in normal cardiovascular development. Based on our studies, we propose that Lgdel genes are required for the expression of soluble signals that regulate neural crest cell differentiation.


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